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rest of your computer, and only the site that created the cookie can read it.Print version ISSN Abstract; ;
. Oculomotor evaluation in patients with peripheral vestibular dysfunction. Rev. Bras. Otorrinolaringol. [online].
vol.72, n.3, pp.407-413.
http://dx.doi.org/10.-00019.AIM: To assess whether or not the parameters of fixed and randomized saccadic movements, of pendular tracking and of optokinetic nystagmus in the digital vectonystagmography may show abnormalities in patients with possible diagnosis of peripheral vestibular dysfunction. METHOD: 60 patients with dizziness of peripheral vestibular origin, from 12 to 82 years of age, males and females, were evaluated in the Universidade Federal de S?o Paulo. Ocular movement parameter findings were compared to a normal pattern. RESULTS: Fixed saccadic movements were altered in 100% of the cases as to latency, and in 35.0% of t the randomized saccadic movements were altered in 100% of the cases as to latency, in 78.3% as to precision, and in 1.7% the pendular tracking showed a gain alteration in the frequencies of 0.1 Hz in 15% of the cases, 0.2 Hz in 21.7%, and 0.4 Hz in 13.3%; the optokinetic nystagmus showed an alteration of the angular speed in the slow component in 1.7% of the cases, and in gain in 5.0%. CONCLUSION: Fixed saccadic movement latency and speed, randomized saccadic movement latency, precision and speed, pendular tracking gain, slow component angular speed, and optokinetic nystagmus gain in the digital vectonystagmography may show abnormalities in patients with possible diagnosis of peripheral vestibular dysfunction.Keywords
ve vertigo.
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Simulations of a mortality plateau in the sexual Penna model for
biological ageing
Abstract: The Penna model is a strategy to simulate the genetic dynamics of
age-structured populations, in which the individuals genomes are represented by
bit-strings. It provides a simple metaphor for the evolutionary process in
terms of the mutation accumulation theory. In its original version, an
individual dies due to inherited diseases when its current number of
accumulated mutations, n, reaches a threshold value, T. Since the number of
accumulated diseases increases with age, the probability to die is zero for
very young ages (n & T) and equals 1 for the old ones (n &= T). Here, instead
of using a step function to determine the genetic death age, we test several
other functions that may or may not slightly increase the death probability at
young ages (n & T), but that decreases this probability at old ones. Our
purpose is to study the oldest old effect, that is, a plateau in the mortality
curves at advanced ages. Imposing certain conditions, it has been possible to
obtain a clear plateau using the Penna model. However, a more realistic one
appears when a modified version, that keeps the population size fixed without
fluctuations, is used. We also find a relation between the birth rate, the
age-structure of the population and the death probability.
Populations and Evolution (q-bio.PE)
[q-bio.PE]
[q-bio.PE] for this version)
Submission history
From: Veit Schwämmle []
[v1] Tue, 22 Feb :33 GMT